Neurology of acute organophosphate poisoning

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Neurology of acute organophosphate poisoning.

Acute organophosphate (OP) poisoning is one of the most common poisonings in emergency medicine and toxicological practice in some of the less-developed nations in South Asia. Traditionally, OP poisoning comes under the domain of emergency physicians, internists, intensivists, and toxicologists. However, some of the complications following OP poisoning are neurological and involve neurologists....

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Cardiovascular Effects of Acute Organophosphate Poisoning

Background:Cardiovascular effects of acute organophosphate (OP) poisoning are common. This study was aimed to assess the cardiovascular effects of OP poisoned patients in Nepal. Methods:This was a prospective hospital-based cross-sectional study of 115 acute OP poisoned patients presenting in emergency department of a tertiary care teaching hospital of central Nepal during November 2008 to Octo...

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Acute pancreatitis induced by acute organophosphate poisoning.

A 32-year-old male with acute organophosphate poisoning developed hyperglycaemia, glycosuria and ketonuria soon after admission to hospital. Serum amylase estimations suggested a diagnosis of acute pancreatitis. He required insulin therapy to control his hyperglycaemia and the organophosphate poisoning was successfully managed by artificial ventilation, an infusion of pralidoxime and intermitte...

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Acute and late complications of organophosphate poisoning.

OBJECTIVE To describe the acute and late complications of organophosphate (OP) poisoning. STUDY DESIGN Case series. PLACE AND DURATION OF STUDY Medicine Department at Peoples Medical College Hospital, Nawabshah, from June 2008 to December 2009. METHODOLOGY A total of 300 patients with organophosphate poisoning admitted to the Medical ICU were included. Baseline investigations included blo...

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Electrophysiological studies in acute organophosphate poisoning.

Electrophysiological studies in suicidal patients with organophosphate poisoning are reported. Patients often developed muscular weakness of variable severity owing to diplorisation block at nicotinic receptors. During such paralysis nerve conduction velocity and distal latencies were normal even in severely paralysed patients. The amplitude of the compound action potential was smaller than in ...

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ژورنال

عنوان ژورنال: Neurology India

سال: 2009

ISSN: 0028-3886

DOI: 10.4103/0028-3886.51277